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Medical Oncology

Medical Oncology

Physician insights on cancer treatment protocols, immunotherapy, targeted therapies, and clinical trial updates.

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How would you treat a patient with metastatic RCC who developed posterior reversible encephalopathy syndrome (PRES) on a TKI-containing regimen?

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Medical Oncology · The University of Texas Health Science Center at San Antonio

PRES is a clinical-radiological diagnosis. The pathology (to my knowledge) is not yet clearly defined though is thought to be the consequence of vascular permeability compromise. There are known triggers of this disease including chemotherapy, auto-immune conditions and VEGF-TKI therapy, and hyperte...

How would you treat a young patient with metastatic colorectal cancer who initially tolerated 12 cycles of FOLFOX + bev with disease progression after cycle 3 of maintenance 5-FU + bev?

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Medical Oncology · Mayo Clinic

TRIBE and TRIBE2 studies showed convincing evidence of triplet chemotherapy FOLFOXIRI/bevacizumab has PFS, and more importantly, OS benefits for patients with metastatic colorectal cancer (mCRC) and now, this regimen has been used in our routine clinical practice more and more frequently especially ...

What features help distinguish thyroid myopathy from immune checkpoint inhibitor-associated myopathy?

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Rheumatology · Yale School of Medicine

Immune checkpoint inhibitors (ICIs) can cause myositis (ICI-myositis). Since ICIs can also induce hypothyroidism, myopathy secondary from hypothyroidism can also be associated with ICI therapy. Different from thyroid myopathy, patients with ICI-myositis barely have myoedema or muscle pseudohypertrop...

For treatment of ITP, what would you add to dexamethasone to achieve the fastest recovery in a patient waiting for a procedure?

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Pediatric Hematology/Oncology · John Hopkins Medicine

I usually use IVIG, particularly if the patient has responded in the past.

How do you approach patients with recurrent grade 3+ neutropenia on IMiD-containing regimens (e.g., VRd or KRd) beyond dose reductions?

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Medical Oncology · Winship Cancer Institute of Emory University

IMiD dosing is a challenge. Let's start with what we know: The FDA approved dose of thalidomide is out of date and the OPTIMUM trial suggests 100 mg PO qHS is the "best" dose (Kropff et al., PMID 22133776) because 400 mg is far too toxic. The phase 1 of Pomalidomide does not demonstrate superiority ...

How would you treat a patient with metastatic renal cell cancer with Crohn's disease after failing available tyrosine kinase inhibitor therapies?

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Medical Oncology · Stanford University Medical Center

It is not an absolute contraindication! It depends on how active the Crohns is; what therapy he is on for the Crohns. Patients on auto immune disorders treated with IO have about a 30% chance of a flare. In RCC, prior to the availability of CPI, monotherapy with sequential TKI's was the standard an...

What chemotherapy do you use with radiation for urothelial carcinoma with squamous differentiation?

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Medical Oncology · University of Washington School of Medicine

In case of primary or dominant urothelial carcinoma with squamous cell features, would approach similar to pure urothelial carcinoma and consider same radio-sensitizing chemo that we are using in S1806 phase III trial. Doses below may not fully reflect S1806 protocol and also depends on the patient ...

How would you approach therapy for BR-refractory Waldenstrom Macroglobulinemia when there is a mixed response to zanabrutinib?

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Hematology · University of California Irvine

It goes without saying this is a “data-free zone.” I would consider extrapolating from CLL and adding venetoclax.

What would you offer postoperatively for a pt with resected 3 separate HPV+ SCC primaries with pN1 disease with ECE who is cisplatin ineligible?

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Medical Oncology · University of Michigan Medical School

In view of this patient having ECS and three primaries (including bilateral disease), I would definitely treat postoperatively with chemotherapy in conjunction with radiation therapy. I still favor a platinum-based regimen in view of this patient's neurological toxicity due to previous cisplatin exp...

What are your recommendations for a patient with metastatic non-mutated lung adenocarcinoma who previously had headaches responsive to prednisone but with negative temporal artery biopsy for GCA?

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Rheumatology · Massachusetts General Hospital

GCA is often a challenging diagnosis to make in the absence of objective findings of halo sign, pathologic evidence of vasculitis, or large vessel vasculitis on imaging. Headache of any type will often respond to prednisone so the first step, in this case, is to determine if the patient truly has GC...